Friday, June 14, 2013

Drug Discovery@nature.com 14 June 2013

Drug Discovery

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TABLE OF CONTENTS

14 June 2013

News
Analysis
Research Highlights
Research & Reviews
Careers


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Nature Genetics
Focus on iCOGS

Nature Genetics is pleased to present the iCOGS print and online Focus advancing our understanding of the genetic susceptibility to three common hormone-related cancers-breast, ovarian and prostate cancers. This collection of 13 papers by the COGS (Collaborative Oncological Gene-environment Study) Consortium is accompanied by editorial essays highlighting and analyzing the main themes of this milestone in genetic epidemiology.

View the Focus for FREE at:
www.nature.com/ng/focus/iCOGS  

Produced with support from 
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News

Top

US clinical-research system in need of review
doi:10.1038/498007a
An imminent rethink is required on the country's approach to government-supported health and pharmaceutical studies.
Full Text

'Basket studies' will hold intricate data for cancer drug approvals
doi:10.1038/nm0613-655
Researchers weave a new trial design on the basis of genetics, rather than cancer type.
Full Text

An audience with Menelas Pangalos
doi:10.1038/nrd4044
Menelas Pangalos, Executive Vice President of Innovative Medicines at AstraZeneca, discusses the company's R&D strategy.
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Patent watch: One valid claim is all that is needed
doi:10.1038/nrd4049
Allergan still has patent protection for its glaucoma drug Combigan (brimonidine and timolol) even though a US court ruled that a patent that describes the combination of brimonidine and timolol is obvious.
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Analysis

Top

What matters most in commercial success: first-in-class or best-in-class?
doi:10.1038/nrd4035
Given that it is common for several companies to be simultaneously pursuing promising new targets, this analysis investigates the question of whether it is better to be first or best in a particular new class of drugs.
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Driving cancer through ErbB3
doi:10.1038/scibx.2013.535
Genentech researchers show that mutations in ERBB3 can drive oncogenesis in a process dependent on the receptor's ability to form a heterodimer with HER2. The findings suggest existing HER2-targeted therapies also could be effective against cancers driven by mutations in ERBB3.
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Research Highlights

Top

Inflammatory disorders: Targeting TRAFs tames inflammation
doi:10.1038/nrd4029
Inhibition of tumour necrosis factor receptor-associated factors (TRAFs) through FBX03 could represent a novel potential anti-inflammatory therapeutic strategy.
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G protein-coupled receptors: Pioneering Frizzled family receptor structure solved
doi:10.1038/nrd4030
A crystal structure of the human Smoothened (SMO) receptor — an essential component of the Hedgehog signalling pathway — bound to an antitumour compound has been solved.
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Analgesia: A new player in neuropathic pain pathogenesis
doi:10.1038/nrd4031
The WNT family of proteins, which are involved in the regulation of cellular processes during nervous system development, have a key role in the pathogenesis of neuropathic pain.
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Research & Reviews

Top

Advances in the development of nucleoside and nucleotide analogues for cancer and viral diseases
doi:10.1038/nrd4010
Although nucleoside analogues have been used clinically for decades, efforts continue to improve response rates and reduce side effects. This article highlights recent progress in the development of new nucleoside and nucleotide analogues for cancer and viral diseases.
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The emerging mutational landscape of G proteins and G-protein-coupled receptors in cancer
doi:10.1038/nrc3521
This article discusses deep sequencing studies indicating that nearly 20% of human tumours harbour mutations in G-protein-coupled receptors (GPCRs) and the implications for GPCRs as therapeutic targets in oncology.
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The good, the bad and the ugly – TFH cells in human health and disease
doi:10.1038/nri3447
An increasing number of studies have highlighted new aspects of the differentiation and function of T follicular helper (TFH) cells in disease, which might represent targets for novel therapeutics.
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Small molecule inhibition of the KRAS–PDEδ interaction impairs oncogenic KRAS signalling
doi:10.1038/nature12205
Interfering with binding of PDEδ to KRAS with small molecules provides a novel opportunity to suppress oncogenic RAS signalling by altering its localization to endomembranes.
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Drug Discovery
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Careers

Top

Career snapshots archive
Career snapshots feature people associated with drug discovery and drug development, with the aim of providing expert insights and advice on a wide range of positions and career paths in this field.
Full Text

nature events
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Find the latest scientific conferences, courses, meetings and symposia on natureevents.com. For event advertising opportunities across the Nature Publishing Group portfolio please contact natureevents@nature.com
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