Thursday, September 20, 2012

SciBX: Science-Business eXchange Contents: September 20 2012, Volume 5 / Issue 37

SciBX: Science-Business eXchange

TABLE OF CONTENTS

September 20 2012, Volume 5 / Issue 37

Analysis

Cover Story
Targets and Mechanisms
Tools

The Distillery: Therapeutics

Autoimmune disease
Cancer
Inflammation
Neurology
Ophthalmic disease
Transplantation
Various

The Distillery: Techniques

Assays and screens
Disease models
Markers
New Partnering Opportunities in Alzheimer's Disease!
A comprehensive interactive dashboard from Relay Technology Management 

Click here for more information on Alzheimer's partnering opportunities.
 
Trade Secrets
A Nature Network blog by BIOENTREPRENEUR

Brought to you by Nature Biotechnology

Access the insights, advice and commentary from scientists and entrepreneurs building biotech sectors around the world.

Join the global dialogue on life science entrepreneurship:
http://blogs.nature.com/trade_secrets
 

Analysis

Cover Story

Top

Drawing a bead on lymphangiogenesis
Tim Fulmer
doi:10.1038/scibx.2012.970
A team of German researchers has developed a high throughput assay to identify compounds that block lymphangiogenesis, a process that promotes the progression and metastasis of solid tumors. Some of the top hits were statins, which the researchers plan to test in preclinical cancer models.
Full Text | PDF

Targets and Mechanisms

Top

Stabilizing RyR2
Lauren Martz
doi:10.1038/scibx.2012.971
Two separate teams have found that preventing calcium ion leak from ryanodine receptors could help treat CNS disorders. Based on these findings, Armgo is developing brain-penetrating versions of its ryanodine receptor–stabilizing compounds for neurological indications.
Full Text | PDF

ASIRTaining a Parkinson's disease target
Lev Osherovich
doi:10.1038/scibx.2012.972
A Tufts University team has compelling evidence that reducing sirtuin 2 activity could be beneficial in Parkinson's disease. The findings argue for developing brain-penetrating SIRT2 inhibitors.
Full Text | PDF

Tools

Top

Strategic synergy
Tracey Baas
doi:10.1038/scibx.2012.973
NYU researchers have shown that the induction of NKG2D ligands contributes to the synergy of radiotherapy with an anti–CTLA-4 mAb. The findings could be used to identify patients most likely to respond to anti–CTLA-4 treatment and to help establish which radiation regimens would enhance the treatment.
Full Text | PDF

Distillery: Therapeutics

Autoimmune disease

Top

Not applicable
doi:10.1038/scibx.2012.974
Mouse studies suggest testosterone could help treat MS.
Full Text | PDF

Cancer

Top

Signal regulatory protein-α (SIRPA)
doi:10.1038/scibx.2012.975
Patient sample and mouse studies suggest disrupting the interaction between SIRPA and CD47 could help treat AML.
Full Text | PDF

Cathepsin B
doi:10.1038/scibx.2012.976
In vitro and mouse studies have identified a bone-targeting doxorubicin prodrug that could help treat bone metastasis.
Full Text | PDF

β-Catenin (CTNNB1); B cell CLL lymphoma 9 (BCL9); wingless-type MMTV integration site (WNT)
doi:10.1038/scibx.2012.977
Mouse studies suggest disrupting the interaction between CTNNB1 and the WNT pathway coactivator BCL9 could help treat WNT-driven cancers.
Full Text | PDF

CXC chemokine receptor 2 (CXCR2; IL8RB)
doi:10.1038/scibx.2012.978
Mouse studies suggest CXCR2 antagonists could help treat cancer.
Full Text | PDF

Ubiquitin specific peptidase 7 (USP7; HAUSP)
doi:10.1038/scibx.2012.979
In vitro and mouse studies suggest the USP7 inhibitor P5091 could help treat MM.
Full Text | PDF

Matrix metalloproteinase 2 (MMP2); MMP9
doi:10.1038/scibx.2012.980
In vitro and mouse studies identified the endohedral metallofullerenol Gd@C82(OH)22 as an MMP inhibitor that could help treat pancreatic cancer.
Full Text | PDF

Protein kinase Cβ (PRKCB)
doi:10.1038/scibx.2012.981
In vitro and mouse studies suggest PRKCB inhibitors could help treat Ewing's sarcoma.
Full Text | PDF

Inflammation

Top

NEDD8 activating enzyme (NAE)
doi:10.1038/scibx.2012.982
Cell culture studies suggest inhibiting neddylation with NAE inhibitors could help treat inflammation.
Full Text | PDF

Bromodomain containing 4 (BRD4)
doi:10.1038/scibx.2012.983

Full Text | PDF

Neurology

Top

EPH receptor A4 (EPHA4)
doi:10.1038/scibx.2012.984
In vitro and in vivo studies suggest inhibiting EPHA4 could help treat ALS.
Full Text | PDF

Sirtuin 2 (SIRT2)
doi:10.1038/scibx.2012.985
Mouse studies suggest antagonizing SIRT2 could be useful for treating PD.
Full Text | PDF

Ophthalmic disease

Top

IL-12; IL-27
doi:10.1038/scibx.2012.986
In vitro and mouse studies suggest a heterodimeric cytokine could help treat uveitis.
Full Text | PDF

Transplantation

Top

Smoothened (SMO)
doi:10.1038/scibx.2012.987
Mouse and patient studies suggest inhibiting SMO to reduce hedgehog pathway signaling could help prevent or treat sclerodermatous GvHD.
Full Text | PDF

Various

Top

Peroxisome proliferation–activated receptor-γ (PPARG; PPARγ)
doi:10.1038/scibx.2012.988
Cell culture studies identified oxygenated polyketide-based PPARγ activators from the Plakinastrella mamillaris sponge that could help treat diabetes and inflammatory diseases.
Full Text | PDF

Distillery: Techniques

Assays and screens

Top

High throughput screen for inhibitors of lymphangiogenesis
doi:10.1038/scibx.2012.989
A high throughput, cell-based assay could be useful for identifying inhibitors of tumor lymphangiogenesis, which is the aberrant growth of the lymphatic system that can drive metastasis of solid cancers.
Full Text | PDF

Silicon-based peptide arrays for mapping antibody-histone interactions
doi:10.1038/scibx.2012.990
A silicon-based peptide array for mapping the interactions of antibodies with differentially modified histones could be used to identify patients who will respond to therapy.
Full Text | PDF

Disease models

Top

Cell line–based culture system for generating human bocavirus
doi:10.1038/scibx.2012.991
A cell line that generates human bocavirus 1 could be useful for developing vaccines and models of bocavirus infection.
Full Text | PDF

Markers

Top

Epigenetic markers to predict risk of long-term liver damage
doi:10.1038/scibx.2012.992
Rat studies suggest heritable epigenetic modifications induced by liver fibrosis could help predict long-term liver damage susceptibility in offspring.
Full Text | PDF

Killer cell lectin-like receptor subfamily K member 1 (KLRK1; CD314; NKG2D) ligands as markers to predict response to anti–CTLA-4 (CD152) therapies and guide selection of radiation therapy regimens
doi:10.1038/scibx.2012.993
Mouse studies suggest levels of NKG2D ligands could be used as a marker to predict patient responsiveness to anti–CTLA-4 therapies and help guide selection of radiation therapy regimens.
Full Text | PDF

Mutations in the extracellular domain of HER2 (EGFR2; ErbB2; neu) as markers of susceptibility to ErbB2 inhibitors
doi:10.1038/scibx.2012.994
In vitro studies identified oncogenic mutations in the ErbB2 extracellular domain and suggest ErbB2 inhibitors could help treat cancers that harbor such mutations.
Full Text | PDF

You have been sent this Table of Contents Alert because you have opted in to receive it. You can change or discontinue your e-mail alerts at any time, by modifying your preferences on your nature.com account at: www.nature.com/myaccount
(You will need to log in to be recognised as a nature.com registrant)

For further technical assistance, please contact our registration department

For print subscription enquiries, please contact our subscription department

For other enquiries, please contact our customer feedback department

Nature Publishing Group | 75 Varick Street, 9th Floor | New York | NY 10013-1917 | USA

Nature Publishing Group's worldwide offices:
London - Paris - Munich - New Delhi - Tokyo - Melbourne
San Diego - San Francisco - Washington - New York - Boston

Macmillan Publishers Limited is a company incorporated in England and Wales under company number 785998 and whose registered office is located at Brunel Road, Houndmills, Basingstoke, Hampshire RG21 6XS.

© 2012 Nature Publishing Group, a division of Macmillan Publishers Limited. All Rights Reserved.

nature publishing group

No comments: