Oncogenesis - Table of Contents alert Volume 1 Issue 7

TABLE OF CONTENTS Volume 1, July 2012 In this issue Original Articles Sign up for e-alerts Web feed Advertisement Nature and Nature Reviews Cancer WEB FOCUS: CANCER METABOLISM A renaissance in the field of cancer metabolism has demonstrated links between oncogenic pathways and the mechanistic basis of tumour cell metabolism. This joint Nature and Nature Reviews Cancer Web Focus highlights recent progress in the field and the promise of therapeutics based on interfering with metabolic pathways. Access the Web Focus at: http://www.nature.com/focus/cancermetabolism   Original Articles Top Genome-wide profiling reveals transcriptional repression of MYC as a core component of NR4A tumor suppression in acute myeloid leukemia S P Boudreaux, A M Ramirez-Herrick, R P Duren and O M Conneely Oncogenesis 2012 1: e19; 10.1038/oncsis.2012.19 Abstract | Full Text Methylation profile of triple-negative breast carcinomas M T Branham, D M Marzese, S R Laurito, F E Gago, J I Orozco, O M Tello, L M Vargas-Roig and M Roqué Oncogenesis 2012 1: e17; 10.1038/oncsis.2012.17 Abstract | Full Text Evaluation of epigenetic modulation of cyclooxygenase-2 as a prognostic marker for hepatocellular carcinoma A Fernández-Alvarez, C Llorente-Izquierdo, R Mayoral, N Agra, L Boscá, M Casado and P Martín-Sanz Oncogenesis 2012 1: e23; 10.1038/oncsis.2012.23 Abstract | Full Text Strong EGFR signaling in cell line models of ERBB2-amplified breast cancer attenuates response towards ERBB2-targeting drugs F Henjes, C Bender, S von der Heyde, L Braun, H A Mannsperger, C Schmidt, S Wiemann, M Hasmann, S Aulmann, T Beissbarth and U Korf Oncogenesis 2012 1: e16; 10.1038/oncsis.2012.16 Abstract | Full Text PEA-15 unphosphorylated at both serine 104 and serine 116 inhibits ovarian cancer cell tumorigenicity and progression through blocking β-catenin J Lee, C Bartholomeusz, S Krishnamurthy, P Liu, H Saso, T A LaFortune, G N Hortobagyi and N T Ueno Oncogenesis 2012 1: e22; 10.1038/oncsis.2012.22 Abstract | Full Text Overexpression of ETV4 is oncogenic in prostate cells through promotion of both cell proliferation and epithelial to mesenchymal transition A Pellecchia, C Pescucci, E De Lorenzo, C Luceri, N Passaro, M Sica, R Notaro and M De Angioletti Oncogenesis 2012 1: e20; 10.1038/oncsis.2012.20 Abstract | Full Text Selective growth inhibition by glycogen synthase kinase-3 inhibitors in tumorigenic HeLa hybrid cells is mediated through NF-κB-dependent GLUT3 expression M Watanabe, N Abe, Y Oshikiri, E J Stanbridge and T Kitagawa Oncogenesis 2012 1: e21; 10.1038/oncsis.2012.21 Abstract | Full Text Natureevents is a fully searchable, multi-disciplinary database designed to maximise exposure for events organisers. The contents of the Natureevents Directory are now live. The digital version is available here. Find the latest scientific conferences, courses, meetings and symposia on natureevents.com. For event advertising opportunities across the Nature Publishing Group portfolio please contact natureevents@nature.com More Nature Events You have been sent this Table of Contents Alert because you have opted in to receive it. You can change or discontinue your e-mail alerts at any time, by modifying your preferences on your nature.com account at: www.nature.com/nams/svc/myaccount (You will need to log in to be recognised as a nature.com registrant). For further technical assistance, please contact our registration department. For print subscription enquiries, please contact our subscription department. For other enquiries, please contact our customer feedback department. Nature Publishing Group | 75 Varick Street, 9th Floor | New York | NY 10013-1917 | USA Nature Publishing Group's worldwide offices: London - Paris - Munich - New Delhi - Tokyo - Melbourne San Diego - San Francisco - Washington - New York - Boston Macmillan Publishers Limited is a company incorporated in England and Wales under company number 785998 and whose registered office is located at Brunel Road, Houndmills, Basingstoke, Hampshire RG21 6XS. © 2012 Nature Publishing Group, a division of Macmillan Publishers Limited. All Rights Reserved.