Oncogene - Table of Contents alert Volume 30 Issue 23

It has come to our attention that the eAlert for Oncogene Volume 30 Issue 23 was compromised. We are resending it now to ensure that everyone who has registered to receive Oncogene eAlerts has received their full complement for the volume. The issue containing all the content went live online as scheduled on June 9, 2011 and has been available since that date to all subscribers. We apologize for any inconvenience this may have caused. TABLE OF CONTENTS Volume 30, Issue 23 (June 2011) In this issue Review Original Articles Short Communication Also new AOP Advertisment Agilent's Complete Cytogenetics Solution Detect more chromosomal aberrations on a single microarray with Agilent's SurePrint G3 CGH+SNP Microarrays. Catalog microarray content which was developed with ISCA or easily add your own custom CGH content to an array. CytoGenomics software helps you make the most of your data, with streamlined analysis and customizable reports. Sign up for e-alerts Recommend to your library Web feed Subscribe Advertisement Scientific Reports – Call for papers Publishing made easy! Entirely open access with papers accepted on technical merit, the community evaluates the importance of papers post-peer review. Scientific Reports exists to facilitate the rapid peer review and publication of research that is of interest to specialists within any given field in the natural sciences. Submit now!   Review Top Nucleophosmin and its complex network: a possible therapeutic target in hematological diseases E Colombo, M Alcalay and P G Pelicci Oncogene 2011 30: 2595-2609; advance online publication, January 31, 2011; 10.1038/onc.2010.646 Abstract | Full Text Original Articles Top α-Catulin knockdown induces senescence in cancer cells L-C Fan, W-F Chiang, C-H Liang, Y-T Tsai, T-Y Wong, K-C Chen, T-M Hong and Y-L Chen Oncogene 2011 30: 2610-2621; advance online publication, January 31, 2011; 10.1038/onc.2010.637 Abstract | Full Text Mutations of the von Hippel–Lindau gene confer increased susceptibility to natural killer cells of clear-cell renal cell carcinoma A Perier, G Fregni, S Wittnebel, S Gad, M Allard, N Gervois, B Escudier, B Azzarone and A Caignard Oncogene 2011 30: 2622-2632; advance online publication, January 24, 2011; 10.1038/onc.2010.638 Abstract | Full Text HEF1, a novel target of Wnt signaling, promotes colonic cell migration and cancer progression Y Li, J H Bavarva, Z Wang, J Guo, C Qian, S N Thibodeau, E A Golemis and W Liu Oncogene 2011 30: 2633-2643; advance online publication, February 14, 2011; 10.1038/onc.2010.632 Abstract | Full Text MicroRNA-451 functions as a tumor suppressor in human non-small cell lung cancer by targeting ras-related protein 14 (RAB14) R Wang, Z-X Wang, J-S Yang, X Pan, W De and L-B Chen Oncogene 2011 30: 2644-2658; advance online publication, February 28, 2011; 10.1038/onc.2010.642 Abstract | Full Text Loss of a single Hic1 allele accelerates polyp formation in ApcΔ716 mice H P Mohammad, W Zhang, H S Prevas, B R Leadem, M Zhang, J G Herman, C M Hooker, D N Watkins, B Karim, D L Huso and S B Baylin Oncogene 2011 30: 2659-2669; advance online publication, February 7, 2011; 10.1038/onc.2010.633 Abstract | Full Text p53-paralog DNp73 oncogene is repressed by IFNα/STAT2 through the recruitment of the Ezh2 polycomb group transcriptional repressor B Testoni, V Schinzari, F Guerrieri, S Gerbal-Chaloin, G Blandino and M Levrero Oncogene 2011 30: 2670-2678; advance online publication, March 14, 2011; 10.1038/onc.2010.635 Abstract | Full Text EAPP: Gatekeeper at the crossroad of apoptosis and p21-mediated cell-cycle arrest P Andorfer and H Rotheneder Oncogene 2011 30: 2679-2690; advance online publication, January 24, 2011; 10.1038/onc.2010.639 Abstract | Full Text Short Communication Top Molecular basis of a novel oncogenic mutation in GNAO1 M Garcia-Marcos, P Ghosh and M G Farquhar Oncogene 2011 30: 2691-2696; advance online publication, February 14, 2011; 10.1038/onc.2010.645 Abstract | Full Text Advertisement Call for papers! Submit your best work in cancer research, including mechanisms of cellular oncogene activation, structure and function of encoded proteins, DNA and RNA tumor viruses and all other aspects of oncogene structure and function. Our online submission process is easy and can assist you in becoming part of this distinguished journal. Visit our Author page to learn about the benefits of publishing in the Journal and for guidelines on how to submit your paper.   Natureevents is a fully searchable, multi-disciplinary database designed to maximise exposure for events organisers. The contents of the Natureevents Directory are now live. The digital version is available here. Find the latest scientific conferences, courses, meetings and symposia on natureevents.com. For event advertising opportunities across the Nature Publishing Group portfolio please contact natureevents@nature.com More Nature Events Please note that you need to be a subscriber or site-licence holder to enjoy full-text access to Oncogene. In order to do so, please purchase a subscription. You have been sent this Table of Contents Alert because you have opted in to receive it. You can change or discontinue your e-mail alerts at any time, by modifying your preferences on your nature.com account at: www.nature.com/nams/svc/myaccount (You will need to log in to be recognised as a nature.com registrant). For further technical assistance, please contact our registration department. For print subscription enquiries, please contact our subscription department. For other enquiries, please contact our customer feedback department. Nature Publishing Group | 75 Varick Street, 9th Floor | New York | NY 10013-1917 | USA Nature Publishing Group's worldwide offices: London - Paris - Munich - New Delhi - Tokyo - Melbourne San Diego - San Francisco - Washington - New York - Boston Macmillan Publishers Limited is a company incorporated in England and Wales under company number 785998 and whose registered office is located at Brunel Road, Houndmills, Basingstoke, Hampshire RG21 6XS. © 2011 Nature Publishing Group, a division of Macmillan Publishers Limited. All Rights Reserved.