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June 2016 Volume 17 Number 6 | |||||||||||||||||||||||||||||||||||||||||||||||
In this issue Research Highlights Reviews
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REVIEWS | Top | ||||||||||||||||||||||||||||||||||||||||||||||
Synaptic AMPA receptor composition in development, plasticity and disease Jeremy M. Henley & Kevin A. Wilkinson p337 | doi:10.1038/nrn.2016.37 AMPA receptor (AMPAR) subunit composition is thought to influence trafficking, but recent findings have challenged previously accepted models for how this might occur. In this Review, Henley and Wilkinson provide an overview of how different receptor subunits affect AMPAR assembly, trafficking and function under normal and pathological conditions. Abstract | Full Text | PDF | |||||||||||||||||||||||||||||||||||||||||||||||
Synaptic mechanisms underlying persistent cocaine craving Marina E. Wolf p351 | doi:10.1038/nrn.2016.39 One of the greatest challenges in treating addiction is preventing relapse during abstinence. In this Review, Marina Wolf discusses rodent models of cocaine craving that reveal the synaptic plasticity that occurs in reward-related brain regions during the abstinence phase. Abstract | Full Text | PDF | Supplementary information | |||||||||||||||||||||||||||||||||||||||||||||||
The neuronal code for number Andreas Nieder p366 | doi:10.1038/nrn.2016.40 Primates have a functional network in frontal and parietal cortices that allows them to quantify the number of elements in a stimulus; that is, its numerosity or cardinality. In this Review, Andreas Nieder examines how neurons in this network process cardinal numbers. Abstract | Full Text | PDF | |||||||||||||||||||||||||||||||||||||||||||||||
The expanding biology of the C9orf72 nucleotide repeat expansion in neurodegenerative disease Aaron R. Haeusler, Christopher J. Donnelly & Jeffrey D. Rothstein p383 | doi:10.1038/nrn.2016.38 A nucleotide repeat expansion (NRE) within the chromosome 9 open reading frame 72 (C9orf72) gene is linked to multiple neurological conditions. Rothstein and colleagues evaluate the evidence indicating that the NRE causes a loss of C9orf72 function or drives toxic gain-of-function mechanisms and consider the cellular defects through which the mutation drives disease pathology. Abstract | Full Text | PDF | Supplementary information | |||||||||||||||||||||||||||||||||||||||||||||||
Erratum: Neural correlates of consciousness: progress and problems Christof Koch, Marcello Massimini, Melanie Boly & Giulio Tononi p395 | doi:10.1038/nrn.2016.61 Full Text | PDF | |||||||||||||||||||||||||||||||||||||||||||||||
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*2014 Journal Citation Report (Thomson Reuters, 2015) |
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