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TABLE OF CONTENTS
| | | | Volume 34, Issue 47 (November 2015) | | In this issue Review Original Articles
Also new AOP | | | | | Advertisement | | What articles have your colleagues been reading?
Take a look at the Oncogene top 10 articles list. These are based purely on downloads from the journal's website over the past 15 days. You may have missed something that could help with your current research project!
Click here to access | | | | | | Review | Top | | Lineage factors and differentiation states in lung cancer progressionW K C Cheung and D X Nguyen Oncogene 2015 34: 5771-5780; advance online publication, March 30, 2015; 10.1038/onc.2015.85 Abstract | Full Text | | Original Articles | Top | | The membrane protein melanoma cell adhesion molecule (MCAM) is a novel tumor marker that stimulates tumorigenesis in hepatocellular carcinomaJ Wang, X Tang, W Weng, Y Qiao, J Lin, W Liu, R Liu, L Ma, W Yu, Y Yu, Q Pan and F Sun Oncogene 2015 34: 5781-5795; advance online publication, March 2, 2015; 10.1038/onc.2015.36 Abstract | Full Text | | | | Reactive oxygen species regulate Smac mimetic/TNFα-induced necroptotic signaling and cell deathB Schenk and S Fulda Oncogene 2015 34: 5796-5806; advance online publication, April 6, 2015; 10.1038/onc.2015.35 Abstract | Full Text | | | | MOZ (MYST3, KAT6A) inhibits senescence via the INK4A-ARF pathwayB N Sheikh, B Phipson, F El-Saafin, H K Vanyai, N L Downer, M J Bird, A J Kueh, R E May, G K Smyth, A K Voss and T Thomas Oncogene 2015 34: 5807-5820; advance online publication, March 16, 2015; 10.1038/onc.2015.33 Abstract | Full Text | | | | Blocking CLEC14A-MMRN2 binding inhibits sprouting angiogenesis and tumour growthP J Noy, P Lodhia, K Khan, X Zhuang, D G Ward, A R Verissimo, A Bacon and R Bicknell Oncogene 2015 34: 5821-5831; advance online publication, March 9, 2015; 10.1038/onc.2015.34 Abstract | Full Text | | | | Nuclear heparanase-1 activity suppresses melanoma progression via its DNA-binding affinityY Yang, C Gorzelanny, A T Bauer, N Halter, D Komljenovic, T Bäuerle, L Borsig, M Roblek and S W Schneider Oncogene 2015 34: 5832-5842; advance online publication, March 9, 2015; 10.1038/onc.2015.40 Abstract | Full Text | | | | The DEAD box protein p68: a crucial regulator of AKT/FOXO3a signaling axis in oncogenesisM Sarkar, V Khare, K K N Guturi, N Das and M K Ghosh Oncogene 2015 34: 5843-5856; advance online publication, March 9, 2015; 10.1038/onc.2015.42 Abstract | Full Text | | | | | | | | | | | Natureevents is a fully searchable, multi-disciplinary database designed to maximise exposure for events organisers. The contents of the Natureevents Directory are now live. The digital version is available here.
Find the latest scientific conferences, courses, meetings and symposia on natureevents.com. For event advertising opportunities across the Nature Publishing Group portfolio please contact natureevents@nature.com | | | | | | | | Please note that you need to be a subscriber or site-licence holder to enjoy full-text access to Oncogene. In order to do so, please purchase a subscription. You have been sent this Table of Contents Alert because you have opted in to receive it. You can change or discontinue your e-mail alerts at any time, by modifying your preferences on your nature.com account at: www.nature.com/nams/svc/myaccount (You will need to log in to be recognised as a nature.com registrant). For further technical assistance, please contact our registration department. For print subscription enquiries, please contact our subscription department. For other enquiries, please contact our customer feedback department. Nature Publishing Group |One New York Plaza, Suite 4500 | New York | NY 10004-1562 | USA Nature Publishing Group's worldwide offices: London - Paris - Munich - New Delhi - Tokyo - Melbourne San Diego - San Francisco - Washington - New York - Boston Macmillan Publishers Limited is a company incorporated in England and Wales under company number 785998 and whose registered office is located at Brunel Road, Houndmills, Basingstoke, Hampshire RG21 6XS. © 2015 Nature Publishing Group, a division of Macmillan Publishers Limited. All Rights Reserved. | | | | |
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